| [1] World Health Organization. Global tuberculosis report 2016. Geneva: World Health Organization, 2016.[2] Fol M, Druszczynska M, Wlodarczyk M,et al. Immune response gene polymorphisms in tuberculosis. Acta Biochim Pol, 2015, 62(4):633-640. [3] Dobler CC, Flack JR, Marks GB. Risk of tuberculosis among people with diabetes mellitus: an Australian nationwide cohort study. BMJ Open, 2012, 2(1):e000666.[4] Wellen KE, Hotamisligil GS. Inflammation, stress, and diabetes. J Clin Invest, 2005, 115(5):1111-1119.[5] Shoelson SE, Lee J, Goldfine AB. Inflammation and insulin resistance. J Clin Invest, 2006, 116(7):1793-1801.[6] Qing YF, Zhou JG, Zhang QB, et al. Association of TLR4 Gene rs2149356 polymorphism with primary gouty arthritis in a case-control study. PLoS One, 2013, 8(5):e64845.[7] Bilan PJ, Samokhvalov V, Koshkina A, et al. Direct and macrophage-mediated actions of fatty acids causing insulin resis-tance in muscle cells. Arch Physiol Biochem, 2009, 115(4): 176-190.[8] 李雨泽, 刘玉琴, 陈向丽,等. 2型糖尿病并发肺结核患者血清中炎症细胞因子水平变化的研究. 中国防痨杂志, 2017, 39(2):145-148.[9] 李雨泽, 李楠, 李晓南,等. 细胞因子与糖尿病并发结核病关系的研究进展. 结核病与肺部健康杂志, 2014, 3(1):64-67.[10] 周利君, 卢水华, 李亮. 糖尿病并发肺结核发病机制的研究进展. 中华传染病杂志, 2013, 31(9):568-570.[11] Wang JY, Lee MC, Shu CC, et al. Optimal duration of anti-TB treatment in patients with diabetes: nine or six months? Chest, 2015, 147(2): 520-528.[12] Karachunskií MA, Komliakova EG, Pospelov LE. Specific features of pulmonary tuberculosis course in patients with insulin-dependent diabetes mellitus in relation to a varying HLA phenotype. Probl Tuberk, 1997,(5): 23-25.[13] Chukanova VP, Sergeev AS, Pospelov LE, et al. Epidemiological and immunogenetic analysis of tuberculosis and diabetes mellitus association. Probl Tuberk, 2000, (4): 11-14.[14] 赵雁林, 端木宏谨,宋长兴. 人类白细胞抗原-DRB1 和 DQB1 基因与肺结核合并糖尿病的相关性分析. 中华结核和呼吸杂志, 2001,24(2): 75-79.[15] 张金风, 吴坤, 李雨泽,等. CC趋化因子配体1基因多态性与2型糖尿病并发肺结核发病风险的关系. 中国糖尿病杂志, 2016, 24(5):398-401.[16] Strohmeier GR, Fenton MJ. Roles of lipoarabinomannan in the pathogenesis of tuberculosis. Microbes Infect, 1999, 1(9):709-717.[17] Kaisho T, Akira S. Toll-like receptor function and signaling. J Allergy Clin Immunol, 2006, 117 (5):979-987.[18] Underhill DM, Ozinsky A, Smith KD, et al. Toll-like receptor-2 mediates mycobacteria-induced proinflammatory signaling in macrophages. Proc Natl Acad Sci U S A, 1999, 96(25):14459-14463.[19] Takeda K, Akira S. TLR signaling pathways (Review). Semin Immunol, 2004, 16(1):3-9.[20] Hotamisligil GS, Arner P, Caro JF, et al. Increased adipose tissue expression of tumor necrosis factor-alpha in human obesity and insulin resistance. J Clin Invest, 1995, 95(5):2409-2415.[21] Dandona P, Aljada A, Bandyopadhyay A. Inflammation: the link between insulin resistance, obesity and diabetes (Review). Trends Immunol, 2004, 25(1):4-7.[22] Hotamisligil GS. Inflammation and metabolic disorders. Nature, 2006, 444(7121):860-867. [23] Lumeng CN, Deyoung SM, Bodzin JL, et al. Increased inflammatory properties of adipose tissue macrophages recruited during diet-induced obesity. Diabetes, 2007, 56(1):16-23. |
京公网安备11010202007215号
Total visitors: Visitors of today: Now online:
This work is licensed under Creative Commons Attribution 3.0 License.
