| ［1］Zhang Y, Heym B, Allen B, et al. The catalase-peroxidase gene and isoniazid resistance of Mycobacterium tuberculosis. Nature,1992,358(6387):591-593.［2］Mokrousov I, Otten T, Filipenko M, et al. Detection of isoniazid-resistant Mycobacterium tuberculosis strains by a multiplex allele-specific PCR assay targeting katG codon 315 variation. J Clin Microbiol,2002,40(7):2509-2512.［3］Banerjee A, Dubnau E, Quemard A, et al. inhA, a gene encoding a target for isoniazid and ethionamide in Mycobacterium tuberculosis. Science,1994,263(5144):227-230.［4］Kim SY, Park YJ, Kim WI, et al. Molecular analysis of isoniazid resistance in Mycobacterium tuberculosis isolates recovered from South Korea. Diagn Microbiol Infect Dis, 2003, 47(3): 497-502.［5］中国防痨协会.结核病诊断细菌学检验规程. 中国防痨杂志,1996,18(1):28-31.［6］van Soolingen D, Hermans PW, de Haas PE, et al. Occurrence and stability of insertion sequences in Mycobacterium tuberculosis complex strains: evaluation of an insertion sequence-dependent DNA polymorphism as a tool in the epidemiology of tuberculosis. J Clin Microbiol,1991,29(11):2578-2586.［7］Musser JM, Kapur V, Williams DL, et al. Characterization of the catalase-peroxidase gene (katG) and inhA locus in isoniazid-resistant and -susceptible strains of Mycobacterium tuberculosis by automated DNA sequencing: restricted array of mutations associated with drug resistance. J Infect Dis,1996,173(1):196-202.［8］Haas WH, Schilke K, Brand J, et al. Molecular analysis of katG gene mutations in strains of Mycobacterium tuberculosis complex from Africa. Antimicrob Agents Chemother, 1997, 41(7): 1601-1603.［9］Mokrousov I, Narvskaya O, Otten T, et al. High prevalence of KatG Ser315Thr substitution among isoniazid-resistant Mycobacterium tuberculosis clinical isolates from northwestern Russia, 1996 to 2001. Antimicrob Agents Chemother,2002,46(5):1417-1424.［10］Marttila HJ, Soini H, Eerola E, et al. A Ser315Thr substitution in KatG is predominant in genetically heterogeneous multidrug-resistant Mycobacterium tuberculosis isolates originating from the St.Petersburg area in Russia. Antimicrob Agents Chemother,1998,42(9):2443-2445.［11］Silva MS, Senna SG, Ribeiro MO, et al. Mutations in katG, inhA, and ahpC genes of Brazilian isoniazid-resistant isolates of Mycobacterium tuberculosis. J Clin Microbiol, 2003, 41(9): 4471-4474.［12］Zhang M, Yue J, Yang YP, et al. Detection of mutations associated with isoniazid resistance in Mycobacterium tuberculosis isolates from China. J Clin Microbiol,2005,43(11):5477-5482.［13］Jiao WW, Mokrousov I, Sun GZ, et al. Molecular characteristics of rifampin and isoniazid resistant Mycobacterium tuberculosis strains from Beijing, China. Chin Med J (Engl), 2007, 120(9): 814-819.［14］Chan RC, Hui M, Chan EW, et al. Genetic and phenotypic characterization of drug-resistant Mycobacterium tuberculosis isolates in Hong Kong. J Antimicrob Chemother, 2007, 59(5): 866-873.［15］Luo T, Zhao M, Li X, et al. Selection of mutations to detect multidrug-resistant Mycobacterium tuberculosis strains in Shanghai, China. Antimicrob Agents Chemother,2010,54(3):1075-1081.［16］菅记涌,王嫩寒,易俊莉,等. MTBDR plus方法快速检测北京地区临床结核分枝杆菌分离株利福平和异烟肼耐药性效果评价.中国防痨杂志, 2010(10):611-614.［17］Lee AS, Tang LL, Lim IH, et al. Lack of clinical significance for the common arginine-to-leucine substitution at codon 463 of the katG gene in isoniazid-resistant Mycobacterium tuberculosis in Singapore. J Infect Dis,1997,176(4):1125-1127.［18］Kurepina NE, Sreevatsan S, Plikaytis BB, et al. Characterization of the phylogenetic distribution and chromosomal insertion sites of five IS6110 elements in Mycobacterium tuberculosis: non-random integration in the dnaA-dnaN region. Tuber Lung Dis,1998,79(1):31-42.［19］van Doorn HR, Kuijper EJ, van der Ende A, et al. The susceptibility of Mycobacterium tuberculosis to isoniazid and the Arg→Leu mutation at codon 463 of katG are not associated. J Clin Microbiol,2001,39(4):1591-1594.［20］Herrera L, Valverde A, Saiz P, et al. Molecular characterization of isoniazid-resistant Mycobacterium tuberculosis clinical strains isolated in the Philippines. Int J Antimicrob Agents, 2004,23(6):572-576.［21］Johnsson K, Froland WA, Schultz PG. Overexpression, purification, and characterization of the catalase-peroxidase KatG from Mycobacterium tuberculosis. J Biol Chem, 1997,272(5): 2834-2840.［22］Rouse DA, DeVito JA, Li Z, et al. Site-directed mutagenesis of the katG gene of Mycobacterium tuberculosis: effects on catalase-peroxidase activities and isoniazid resistance. Mol Microbiol, 1996,22(3):583-592. |
京公网安备11010202007215号
Total visitors: Visitors of today: Now online:
This work is licensed under Creative Commons Attribution 3.0 License.
